Afrobeats V1.0 AU VSTi WiN OSX

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Afrobeats V1.0 AU VSTi WiN OSX





 
 
 
 
 
 
 

Afrobeats V1.0 AU VSTi WiN OSX

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StudioLinked Afrobeats v1.0 AU VSTi [Win/Mac]

StudioLinked Afrobeats v1.0 AU VSTi [Win/Mac]

StudioLinked Afrobeats v1.0 AU VSTi [Win/Mac]

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Wistar rats were used to evaluate the effect of orally administered amlodipine on angiogenesis in the aortic ring assay. Among many calcium antagonists, amlodipine showed the most potent inhibitory effect on angiogenesis. Oral administration of 1 mg/kg amlodipine for 10 days suppressed by 38% new blood vessel formation. When the minimum dose that produced a 50% inhibition was determined to be 1 mg/kg, amlodipine inhibited angiogenesis by 51% and by 75% at a dose of 5 and 10 mg/kg, respectively. Amlodipine also decreased the rate of VEGF-induced proliferation of human umbilical vein endothelial cells (HUVECs) by 23% at 1 nM. In the aortic ring assay, 10 nM of amlodipine inhibited vascular development by 50%. Aortic ring sprouting of HUVECs was also suppressed by a 2.5-fold higher concentration of amlodipine, 5.0 microM. In contrast, the calcium channel blocker, nifedipine, did not have such an effect on either VEGF-induced proliferation of HUVECs or on angiogenesis in the aortic ring. Amlodipine decreased the numbers of proliferating and migrating HUVECs by approximately 40%. These results indicate that amlodipine is a potent inhibitor of angiogenesis at therapeutically relevant concentrations. In
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